Search results for "Genetic predisposition"

showing 10 items of 758 documents

Antagonistic effects of a Mhc class I allele on malaria-infected house sparrows.

2008

8 pages; International audience; Genes of the Major Histocompatibility Complex (Mhc) play a fundamental role during the immune response because MHC molecules expressed on cell surface allow the recognition and presentation of antigenic peptides to T-lymphocytes. Although Mhc alleles have been found to correlate with pathogen resistance in several host-parasite systems, several studies have also reported associations between Mhc alleles and an accrued infection risk or an accelerated disease progression. The existence of these susceptibility alleles is puzzling, as the cost generated by the infection should rapidly eliminate them from the population. Here, we show that susceptibility alleles…

0106 biological sciencesPlasmodiumMESH : Molecular Sequence DataMESH : DNAGenes MHC Class IMESH: Amino Acid Sequenceco-evolutionMESH: Base SequenceMESH : Microsatellite Repeats01 natural sciencessusceptibilityMESH: SparrowsPleiotropy[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisMESH: AnimalsMESH : Malaria AvianGenetics0303 health scienceseducation.field_of_studybiologyMESH : Amino Acid Sequence[SDV.BID.EVO]Life Sciences [q-bio]/Biodiversity/Populations and Evolution [q-bio.PE]MESH: DNAMESH: Genetic Predisposition to DiseaseMESH: Genes MHC Class I3. Good healthMESH: Malaria Avian[ SDV.BID.EVO ] Life Sciences [q-bio]/Biodiversity/Populations and Evolution [q-bio.PE]MESH: Haemosporidaavian malariaSparrows[ SDV.MP.PAR ] Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyMalaria AvianMolecular Sequence DataPopulationMESH: Genetics PopulationMajor histocompatibility complex010603 evolutionary biologyMESH : Genes MHC Class Iresistance03 medical and health sciencesImmune systemAvian malariaMHC class ImedicinePasser domesticusAnimalsGenetic Predisposition to Disease[SDV.MP.PAR]Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyAmino Acid SequenceAlleleeducationAllelesEcology Evolution Behavior and Systematics030304 developmental biologyparasite competitionMESH: Molecular Sequence DataBase Sequencehouse sparrowMESH: PlasmodiumMESH: Alleles[ SDV.GEN.GA ] Life Sciences [q-bio]/Genetics/Animal geneticsDNAHaemosporidamedicine.diseaseMESH : Genetics PopulationHistocompatibility[SDV.GEN.GA]Life Sciences [q-bio]/Genetics/Animal geneticsGenetics PopulationMESH : PlasmodiumImmunologybiology.proteinMESH : Base SequenceMESH : Genetic Predisposition to DiseaseAntagonistic pleiotropyMESH : SparrowsMESH : AnimalsMESH : HaemosporidaMESH: Microsatellite RepeatsMESH : AllelesMicrosatellite Repeats[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/Symbiosis
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IL10 promoter haplotypes may contribute to altered cytokine expression and systemic inflammation in celiac disease

2018

Celiac disease (CD) is an autoimmune/inflammatory condition triggered by dietary gluten intake in genetically predisposed individuals. Though associations with MHC class II HLA-DQ2 or -DQ8 are the primary and necessary genetic predisposition for CD, >97% of genetically predisposed individuals never develop CD. Cytokines were measured in the serum of CD patients and controls. Possible associations with IL10 promoter variants were investigated. Cytokine expression from PBMCs was monitored in response to gluten exposure, or CD3/TCR complex stimulation in the absence or presence of recombinant IL-10. Serum cytokines varied between patients with CD at the time of diagnosis, after dietary elimina…

0301 basic medicineAdolescentGenotypeGlutensCD3medicine.medical_treatmentImmunologySystemic inflammationPolymorphism Single NucleotidePeripheral blood mononuclear celllaw.invention03 medical and health sciences0302 clinical medicinelawGenetic predispositionmedicineHumansImmunology and AllergyGenetic Predisposition to DiseaseChildPromoter Regions GeneticInflammationchemistry.chemical_classificationbiologybusiness.industryInterleukin-17GlutenInterleukin-10Celiac DiseaseInterleukin 10030104 developmental biologyCytokineHaplotypeschemistryChild PreschoolImmunologybiology.proteinRecombinant DNACytokines030211 gastroenterology & hepatologymedicine.symptombusinessClinical Immunology
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TLR1 and PRKAA1 Gene Polymorphisms in the Development of Atrophic Gastritis and Gastric Cancer.

2018

Background & Aims: Previous genome-wide association studies showed that genetic polymorphisms in toll-like receptor 1 (TLR1) and protein kinase AMP-activated alpha 1 catalytic subunit (PRKAA1) genes were associated with gastric cancer (GC) or increased Helicobacter pylori (H. pylori) infection susceptibility. The aim of this study was to evaluate the association between TLR1 and PRKAA1 genes polymorphisms and H.pylori infection, atrophic gastritis (AG) or GC in the European population.Methods: Single-nucleotide polymorphisms (SNPs) were analysed in 511 controls, 340 AG patients and 327 GC patients. TLR1 C>T (rs4833095) and PRKAA1 C>T (rs13361707) were genotyped by the real-time po…

0301 basic medicineAdultGastritis AtrophicMalemedicine.medical_specialtyAtrophic gastritisSingle-nucleotide polymorphismAMP-Activated Protein KinasesGastroenterologyPolymorphism Single NucleotideWhite Peoplelaw.inventionHelicobacter Infections03 medical and health sciences0302 clinical medicineGene FrequencylawRisk FactorsStomach NeoplasmsInternal medicineGenotypemedicineSNPHumansGenetic Predisposition to DiseaseAllelePolymerase chain reactionGenetic Association StudiesGenetic associationAgedbiologyHelicobacter pyloribusiness.industryGastroenterologyHelicobacter pyloriMiddle Agedbiology.organism_classificationmedicine.diseaseToll-Like Receptor 1Europe030104 developmental biologyPhenotype030220 oncology & carcinogenesisCase-Control StudiesFemalebusinessJournal of gastrointestinal and liver diseases : JGLD
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Autosomal recessive hypercholesterolemia in Spain.

2017

Abstract Background and aims Autosomal recessive hypercholesterolemia (ARH) is a very rare disease, caused by mutations in LDL protein receptor adaptor 1 (LDLRAP1). It is characterized by high levels of low-density lipoprotein cholesterol (LDL-C) and increased risk of premature cardiovascular disease. We aimed to characterize ARH in Spain. Methods Data were collected from the Dyslipidemia Registry of the Spanish Atherosclerosis Society. A literature search was performed up to June 2017, and all diagnostic genetic studies for familial hypercholesterolemia of Spain were reviewed. Results Seven patients with ARH were identified, 6 true homozygous and one compound heterozygous with a novel muta…

0301 basic medicineAdultGenetic MarkersMalemedicine.medical_specialtyHeterozygoteHypercholesterolemiaDiseaseFamilial hypercholesterolemia030204 cardiovascular system & hematologyCompound heterozygosity03 medical and health sciences0302 clinical medicineInternal medicinemedicinePrevalenceHumansGenetic Predisposition to DiseaseRegistriesChildAdaptor Proteins Signal TransducingHypolipidemic Agentsbusiness.industryGenetic heterogeneityHomozygoteInfantCholesterol LDLMiddle Agedmedicine.diseaseAtherosclerosisUp-Regulation030104 developmental biologyEndocrinologyPhenotypeAutosomal Recessive HypercholesterolemiaSpainChild PreschoolCohortMutationDisease ProgressionFemaleCardiology and Cardiovascular MedicinebusinessDyslipidemiaRare diseaseAtherosclerosis
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Genome-wide association meta-analysis highlights light-induced signaling as a driver for refractive error

2018

Skin affections after sulfur mustard (SM) exposure include erythema, blister formation and severe inflammation. An antidote or specific therapy does not exist. Anti-inflammatory compounds as well as substances counteracting SM-induced cell death are under investigation. In this study, we investigated the benzylisoquinoline alkaloide berberine (BER), a metabolite in plants like berberis vulgaris, which is used as herbal pharmaceutical in Asian countries, against SM toxicity using a well-established in vitro approach. Keratinocyte (HaCaT) mono-cultures (MoC) or HaCaT/THP-1 co-cultures (CoC) were challenged with 100, 200 or 300 mM SM for 1 h. Post-exposure, both MoC and CoC were treated with 1…

0301 basic medicineAdultMaleCell typeResearchInstitutes_Networks_Beacons/MICRAIn silicotaittovirheetGenome-wide association studyRetinal Pigment EpitheliumBiologyBlindnessPolymorphism Single NucleotideSensory disorders Donders Center for Medical Neuroscience [Radboudumc 12]ArticleRetinaWhite People03 medical and health sciencesHIGH-GRADE MYOPIA ; RETINAL-PIGMENT EPITHELIUM ; SEROTONIN PATHWAY GENES ; FORM-DEPRIVATION MYOPIA ; COMMON VARIANTS ; OCULAR GROWTH ; RETINITIS-PIGMENTOSA ; GENOTYPE IMPUTATION ; MISSENSE MUTATIONS ; DOPAMINE-RECEPTORSAsian Peoplerefractive errorsRetinitis pigmentosaGeneticsmedicineMyopiaJournal ArticleHumansGenetic Predisposition to Disease610 Medicine & healthRegulation of gene expressionRetinaRetinal pigment epitheliummedicine.diseaseRefractive Errors030104 developmental biologymedicine.anatomical_structureManchester Institute for Collaborative Research on AgeingGene Expression Regulationgenetic factorsEye disorderFemalesense organsgeneettiset tekijätNeuroscienceGenome-Wide Association StudySignal Transduction
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Mitochondrial DNA haplogroups J and T increase the risk of glioma.

2021

The presence of different sets of mitochondrial polymorphisms generated by the accumulation of mutations in different maternal lineages has allowed differentiating mitochondrial haplogroups in human populations. These polymorphisms, in turn, may have effects at the phenotypic level, considering a possible contribution of these germinal mutations to the development of certain diseases such as cancer. The main goal of the present study is to establish a possible association between mitochondrial haplogroups and the risk of suffering glioma. Blood samples were obtained from 32 patients from Catalonia (Spain) diagnosed with different grades of glioma (II, III and IV), according to the World Hea…

0301 basic medicineAdultMaleMitochondrial DNAPopulationBiologyDNA MitochondrialHaplogroup03 medical and health sciences0302 clinical medicineGliomamedicineHumansGenetic Predisposition to DiseaseeducationMolecular BiologyGeneticseducation.field_of_studyBrain NeoplasmsCancerCell BiologyGliomamedicine.diseasePhenotype030104 developmental biologyIncreased riskHaplotypesMolecular MedicineFemale030217 neurology & neurosurgeryHuman mitochondrial DNA haplogroupMitochondrion
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Genetics of Wilson disease and Wilson-like phenotype in a clinical series from eastern Spain.

2019

Wilson's disease (WD) is an autosomal recessive disorder caused by ATP7B mutations. Subjects with only one mutation may show clinical signs and individuals with biallelic changes may remain asymptomatic. We aimed to achieve a conclusive genetic diagnosis for 34 patients clinically diagnosed of WD. Genetic analysis comprised from analysis of exons to WES (whole exome sequencing), including promoter, introns, UTRs (untranslated regions), besides of study of large deletions/duplications by MLPA (multiplex ligation-dependent probe amplification). Biallelic ATP7B mutations were identified in 30 patients, so that four patients were analyzed using WES. Two affected siblings resulted to be compound…

0301 basic medicineAdultMaleNerve Tissue Proteins030105 genetics & heredityBiologymedicine.disease_causeCompound heterozygosityGenetic analysis03 medical and health sciencesExonHepatolenticular DegenerationExome SequencingGeneticsmedicineHumansGenetic Predisposition to DiseaseMultiplex ligation-dependent probe amplificationGenetic TestingGenetics (clinical)Exome sequencingGeneticsMutationExonsmedicine.diseaseWilson's disease030104 developmental biologyPhenotypeCopper-Transporting ATPasesSpainMutationFemaleCongenital disorder of glycosylationClinical geneticsREFERENCES
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Molecular diagnosis of PIK3CA-related overgrowth spectrum (PROS) in 162 patients and recommendations for genetic testing.

2017

Postzygotic activating mutations of PIK3CA cause a wide range of mosaic disorders collectively referred to as PIK3CA-related overgrowth spectrum (PROS). We describe the diagnostic yield and characteristics of PIK3CA sequencing in PROS. We performed ultradeep next-generation sequencing (NGS) of PIK3CA in various tissues from 162 patients referred to our clinical laboratory and assessed diagnostic yield by phenotype and tissue tested. We identified disease-causing mutations in 66.7% (108/162) of patients, with mutant allele levels as low as 1%. The diagnostic rate was higher (74%) in syndromic than in isolated cases (35.5%; P = 9.03 × 10−5). We identified 40 different mutations and found stro…

0301 basic medicineAdultMalePathologymedicine.medical_specialtyAdolescentGenotypeClass I Phosphatidylinositol 3-KinasesPrenatal diagnosisBioinformaticsmedicine.disease_causeDNA sequencing03 medical and health sciencesYoung Adult0302 clinical medicinePrenatal DiagnosisGenotypemedicineHumansGenetic Predisposition to DiseaseGenetic TestingAlleleChildGenetics (clinical)AllelesGenetic Association StudiesGrowth DisordersGenetic testingMutationmedicine.diagnostic_testbusiness.industryMosaicismInfant NewbornDisease ManagementHigh-Throughput Nucleotide SequencingInfantSequence Analysis DNAPhenotype030104 developmental biologyPhenotypeAmino Acid SubstitutionChild PreschoolMutationAllelic heterogeneityFemalebusiness030217 neurology & neurosurgeryGenetics in medicine : official journal of the American College of Medical Genetics
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Relevance of secretor status genotype and microbiota composition in susceptibility to rotavirus and norovirus infections in humans

2017

Host genetic factors, such as histo-blood group antigens (HBGAs), are associated with susceptibility to norovirus (NoV) and rotavirus (RV) infections. Recent advances point to the gut microbiome as a key player necessary for a viral pathogen to cause infection. In vitro NoV attachment to host cells and resulting infections have been linked to interactions with certain bacterial types in the gut microbiota. We investigated the relationship between host genotype, gut microbiota, and viral infections. Saliva and fecal samples from 35 adult volunteers were analysed for secretor status genotype, the gut microbiota composition by 16S rRNA gene sequencing, and salivary IgA titers to NoV and RV. Hi…

0301 basic medicineAdultMaleSalivaGenotype030106 microbiologyResistanceMicrobiologiaGut floraHuman gut microbiomemedicine.disease_causeArticleRotavirus InfectionsMicrobiologyAssociation03 medical and health sciencesFecesfluids and secretionsBlood group antigensFut2 geneRotavirusGenotypemedicineHumansGenetic Predisposition to DiseaseDiseaseMicrobiomePolymorphismSalivaPathogenEcosystemCaliciviridae InfectionsMultidisciplinaryEnteric bacteriabiologyRuminococcusMiddle Agedbiology.organism_classificationFucosyltransferasesVirusGastrointestinal MicrobiomeGastroenteritis030104 developmental biologyImmunologyNorovirusFemale
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KIR2DL3 and the KIR ligand groups HLA-A-Bw4 and HLA-C2 predict the outcome of hepatitis B virus infection.

2017

Killer immunoglobulin-like receptors (KIRs) regulate the activation of Natural Killer cells through their interaction with human leukocyte antigens (HLA). KIR and HLA loci are highly polymorphic and certain HLA-KIR combinations have been found to protect against viral infections. In this study we analyzed whether the KIR/HLA repertoire may influence the course of hepatitis B virus (HBV) infection. Fifty-seven subjects with chronic hepatitis B (CHB), 44 subjects with resolved HBV infection, and 60 healthy uninfected controls (HC) were genotyped for KIR and their HLA ligands. The frequency of the HLA-A-Bw4 ligand group was higher in CHB (58%) than subjects with resolved infection (23%) (crude…

0301 basic medicineAdultMaleSettore MED/09 - Medicina InternaGenotypeSettore MED/17 - Malattie InfettiveKIR LigandHuman leukocyte antigenHLA-C AntigensBiologymedicine.disease_cause03 medical and health sciencesYoung Adult0302 clinical medicineHepatitis B ChronicVirologymedicineHumansGenetic Predisposition to DiseaseAlleleReceptorGeneAgedSettore MED/04 - Patologia GeneraleHepatitis B virusHepatologyHLA-A AntigensMiddle AgedVirologyHLA-AKIRs HLAA HBV030104 developmental biologyInfectious DiseasesTreatment OutcomeHLA-B AntigensReceptors KIR2DL3ImmunologyFemalePredictive variables030215 immunology
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